浜松医科大学教員総覧教員プロフィール

論文

[1]. Static magnetic field stimulation enhances shunting inhibition via a SLC26 family Cl⁻channel, inducing intrinsic plasticity
The Journal of Neuroscience Society for Neuroscience [巻]44 [号]9 [頁]e1324222024 （2024年） [査読] 有 [DOI]

[2]. 新カリキュラム施行から1年　看護基礎教育の今を探る　Part３　新年度から新たに取り組む科目の展望　「アクティブラーナーを育てる」
看護展望メヂカルフレンド社 [巻]48 [号]4 [頁]148 -152 （2023年） [査読] 無

[3]. A novel de novo KCNB1 variant altering channel characteristics in a patient with periventricular heterotopia, abnormal corpus callosum, and mild seizure outcome.
Journal of Human Genetics Springer Nature [巻]68 [号]1 [頁]25 -31 （2023年） [査読] 有 [DOI]

[4]. Taurine depletion during fetal and postnatal development blunts firing responses of neocortical layer II/III pyramidal neurons.
Frontiers in Molecular Neuroscience [巻]15 [頁]806798 （2022年） [査読] 有 [DOI]

[5]. WNK3 kinase maintains neuronal excitability by reducing inwardly rectifying K⁺ conductance in layer V pyramidal neurons of mouse medial prefrontal cortex.
Frontiers in Molecular Neuroscience [巻]15 [頁]856262 （2022年） [査読] 有 [DOI]

[6]. ATP6V0A1 encoding the a1-subunit of the V0 domain of vacuolar H⁺-ATPases is essential for brain development in humans and mice.
Nature Communications [巻]12 [号]1 [頁]2107 （2021年） [査読] 有 [DOI]

[7]. 細胞内タウリンのプロテインキナーゼCによるリン酸化を介したGABA_A受容体膜発現制御
タウリンリサーチ国際タウリン研究会 [巻]6 [号]1 [頁]20 -22 （2020年） [査読] 有

[8]. タウリントランスポーター(TauT)欠損の大脳皮質II/III層錐体神経発火活動への影響について
タウリンリサーチ国際タウリン研究会 [巻]6 [号]1 [頁]18 -19 （2020年） [査読] 有

[9]. Intracellular Cl^- dysregulation causing and caused by pathogenic neuronal activity.
Pflűgers Archiv : European Journal of Physiology [巻]472 [号]7 [頁]977 -987 （2020年） [査読] 有 [DOI] [機関リポジトリ]

[10]. Alterations of GABAergic Neuron-Associated Extracellular Matrix and Synaptic Responses in Gad1-Heterozygous Mice Subjected to Prenatal Stress.
Frontiers in Cellular Neuroscience [巻]12 [頁]284 （2018年） [査読] 有 [DOI]

[11]. De novo variants in CAMK2A and CAMK2B cause neurodevelopmental disorders.
Annals of Clinical and Translational Neurology [巻]5 [号]3 [頁]280 -296 （2018年） [査読] 有 [DOI]

[12]. Biallelic Variants in CNPY3, Encoding an Endoplasmic Reticulum Chaperone, Cause Early-Onset Epileptic Encephalopathy.
American Journal of Human Genetics [巻]102 [号]2 [頁]321 -329 （2018年） [査読] 有 [DOI]

[13]. Impaired neuronal KCC2 function by biallelic SLC12A5 mutations in migrating focal seizures and severe developmental delay.
Scientific Reports [巻]6 [頁]30072 （2016年） [査読] 有 [DOI]

[14]. Ion channels, guidance molecules, intracellular signaling and transcription factors regulating nervous and vascular system development
The Journal of Physiological Sciences [巻]66 [号]2 [頁]175 -188 （2015年） [査読] 有 [URL] [DOI]

[15]. De novo KCNB1 mutations in infantile epilepsy inhibit repetitive neuronal firing.
Scientific Reports [巻]5 [頁]15199 （2015年） [査読] 有 [URL] [DOI]

[16]. Characteristics and roles of the volume-sensitive outwardly rectifying (VSOR) anion channel in the central nervous system.
Neuroscience [巻]275 [頁]211 -231 （2014年） [査読] 有 [URL] [DOI]

[17]. Roles of taurine-mediated tonic GABA_A receptor activation in the radial migration of neurons in the fetal mouse cerebral cortex.
Frontiers in Cellular Neuroscience [巻]8 [頁]88 （2014年） [査読] 有 [URL] [DOI]

[18]. A Newly Cloned ClC-3 Isoform, ClC-3d, as well as ClC-3a Mediates Cd⁺-Sensitive Outwardly Rectifying Anion Currents.
Cellular Physiology and Biochemistry [巻]33 [号]3 [頁]539 -556 （2014年） [査読] 有 [URL] [DOI]

[19]. Activity-dependent endogenous taurine release facilitates excitatory neurotransmission in the neocortical marginal zone of neonatal rats.
Frontiers in Cellular Neuroscience [巻]8 [頁]33 （2014年） [査読] 有 [URL] [DOI]

[20]. De novo mutations in GNAO1, encoding a Gα_o subunit of heterotrimeric G proteins, cause epileptic encephalopathy.
American Journal of Human Genetics [巻]93 [号]3 [頁]496 -505 （2013年） [査読] 有 [URL] [DOI]

[21]. Early-phase occurrence of K⁺ and Cl^- efflux in addition to Ca²⁺ mobilization is a prerequisite to apoptosis in HeLa cells.
Apoptosis [巻]17 [号]8 [頁]821 -831 （2012年） [査読] 有 [URL] [DOI]

[22]. Ca²⁺ nanodomain-mediated component of swelling-induced volume-sensitive outwardly rectifying anion current triggered by autocrine action of ATP in mouse astrocytes.
Cellular Physiology and Biochemistry [巻]28 [号]6 [頁]1181 -1190 （2011年） [査読] 有 [URL] [DOI]

[23]. Regulation of bradykinin-induced activation of volume-sensitive outwardly rectifying anion channels by Ca²⁺ nanodomains in mouse astrocytes.
The Journal of Physiology [巻]589 [号]16 [頁]3909 -3927 （2011年） [査読] 有 [URL] [DOI]

[24]. Calcium dependence of the priming, activation and inactivation of ryanodine receptors in frog motor nerve terminals.
European Journal of Neuroscience [巻]32 [号]6 [頁]948 -962 （2010年） [査読] 有 [URL] [DOI]

[25]. Bradykinin-induced astrocyte-neuron signalling: glutamate release is mediated by ROS-activated volume-sensitive outwardly rectifying anion channels.
The Journal of Physiology [巻]587 [号]10 [頁]2197 -2209 （2009年） [査読] 有 [DOI]

[26]. Ca²⁺-dependent inactivation of Ca²⁺-induced Ca²⁺ release in bullfrog sympathetic neurons.
The Journal of Physiology [巻]586 [号]14 [頁]3365 -3384 （2008年） [査読] 有 [DOI]

[27]. Overexpression of human CD38/ADP-ribosyl cyclase enhances acetylcholine-induced Ca²⁺ signalling in rodent NG108-15 neuroblastoma cells.
Neuroscience Research [巻]57 [号]3 [頁]339 -346 （2007年） [査読] 有 [DOI]

[28]. Adenosine depresses a Ca²⁺-independent step in transmitter exocytosis at frog motor nerve terminals.
European Journal of Neuroscience [巻]15 [号]8 [頁]1291 -1298 （2002年） [査読] 有 [URL] [DOI]

[29]. Ca²⁺-dependent Ca²⁺ clearance via mitochondrial uptake and plasmalemmal extrusion in frog motor nerve terminals.
Journal of Neurophysiology [巻]87 [号]4 [頁]1816 -1823 （2002年） [査読] 有 [URL] [DOI]

[30]. Functional triads consisting of ryanodine receptors, Ca²⁺ channels, and Ca²⁺-activated K⁺ channels in bullfrog sympathetic neurons: Plastic modulation of action potential.
Journal of General Physiology [巻]116 [号]5 [頁]697 -720 （2000年） [査読] 有 [URL] [DOI]

[31]. Functional coupling of Ca²⁺ channels to ryanodine receptors at presynaptic terminals: Amplification of exocytosis and plasticity.
Journal of General Physiology [巻]115 [号]4 [頁]519 -532 （2000年） [査読] 有 [URL] [DOI]

[32]. シナプス前終末の細胞内Ca²⁺遊離とシナプスの可塑性
蛋白質核酸酵素共立出版 [巻]45 [号]3 Suppl [頁]498 -503 （2000年） [査読] 無

[33]. シナプス前終末におけるCICRの測定
臨床神経科学中外医学社 [巻]17 [号]8 [頁]10 -11 （1999年） [査読] 無

[34]. Long-term use-dependent enhancement of impulse-induced exocytosis by adrenaline at frog motor nerve terminals.
Neuroscience Research [巻]33 [頁]239 -244 （1999年） [査読] 有

[35]. A Ca²⁺-induced Ca²⁺ release mechanism involved in asynchronous exocytosis at frog motor nerve terminals.
Journal of General Physiology [巻]112 [号]5 [頁]593 -609 （1998年） [査読] 有 [URL] [DOI]