Hamamatsu University Hospital Pediatrics
Senior Assistant Professor
藤澤 泰子
FUJISAWA Yasuko

e-mail:yasuko@@@hama-med.ac.jp (* please replace "@@@" to "@")

Last updated: 2026/05/08
Basic information on teaching staff
Profile(Japanese)
Graduated from Hamamatsu University School of Medicine in March 1998 and obtained a medical license in the same year. Engaged in clinical practice in general pediatrics, with a particular specialization in pediatric endocrinology. Conducts research on DOHaD (Developmental Origins of Health and Disease), especially using animal models to investigate the association between fetal undernutrition and postnatal male reproductive disorders. Has also published multiple reports on rare diseases in the field of pediatric endocrinology. In addition, conducts research on steroid biosynthesis.
Career
1998/4/- 浜松医科大学
1999/2/- 聖隷沼津病院
2007/5/- カリフォルニア大学デービス校
2010/4/- 松医科大学医学部付属病院
2013/8/- 浜松医科大学
2016/4/- 浜松医科大学
Licenses and qualifications
[1]. 医師免許
Current state of research and teaching activities
Provides clinical care in general pediatrics and pediatric endocrinology. Research activities focus primarily on the field of DOHaD (Developmental Origins of Health and Disease), a concept proposing that future health and susceptibility to specific diseases are strongly influenced by the fetal and early postnatal environment. Also serves as a committee member of the Women Physicians Support Center at Hamamatsu University School of Medicine, where she is involved in initiatives to support the continued career development of female physicians.
Research Areas
Life Science - Metabolism and endocrinology
Life Science - Embryonic medicine and pediatrics
Research keywords
pediatrics endocrinology molecular genetics sex differentiation DOHaD steroido biogenesis
Subject of research
[1]. DOHaD Testicular dysgenesis and fetal environment Steroid genesis in organs
Research Projects (Competitive Research Funds)
[1]. 先端脳科学研究と光量子医学の融合による精神神経疾患・診断治療薬創出と若手医学研究者活躍プラットフォームの構築
[2]. 精巣体細胞栄養代謝プログラミング機構の解明
[3]. 代謝に制御されるエピゲノムに着目した精巣形成不全症発症の分子基盤の解明
[4]. 胎生期の低栄養環境による精巣機能障害の発症機序の解明
[5]. 母乳栄養による肥満発症抑制機序〜脂肪細胞と腸管内分泌細胞への複合作用の解明
[6]. 母乳に含まれる抗肥満生理活性物質の探索
Research activities
Papers
[1]. FIGLAホモ接合性ミスセンスバリアントにより早発卵巣不全を発症した姉妹例
日本小児遺伝学会学術集会プログラム・抄録集 [Volume]47th (2026/) [Reviewed] No
[2]. Novel HK1 intronic variant in congenital hyperinsulinism: impaired transactivation function for FOXA2.
European journal of endocrinology (2026/) [Reviewed] Yes
[3]. De novo GNAS-Gsα variant (p.Thr55Ala) with constitutive gain-of-function effects on AVPR2 and PTH1R signalings.
Journal of human genetics (2026/) [Reviewed] Yes [DOI]
[4]. Comprehensive Molecular Studies in 88 Japanese Patients With Congenital Hypogonadotropic Hypogonadism.
The Journal of clinical endocrinology and metabolism (2025/) [Reviewed] Yes
[5]. Homozygous missense variant in two Japanese sisters with primary ovarian insufficiency: Case reports and literature review.
Reproductive medicine and biology [Volume]24 [Issue]1 [Page]e12635 (2025/) [Reviewed] Yes
Research presentations
[1]. 2025/10/29 小児副腎皮質腫瘍における11-ketotestosteron(11-KT)産生とそのメカニズムの解析 第32回日本ステロイドホルモン学会学術集会 
[2]. 2024/10/10 Year book 副腎 第57回日本小児内分泌学会学術集会
[3]. 2024/6/7 エストロゲン受容体αをコードするESR1遺伝子イントロン6の微細欠失は停留精巣・尿道下裂の発症感受性因子である 第97回日本内分泌学会
[4]. 2024/6/7 「内分泌疾患を診療する内科医・産婦人科医・泌尿器科医・小児科医のための性分化疾患(DSD)の診療ガイドライン2024」エビデンスに基づく小児期DSDの診断・診療アプローチ
[5]. 2023/2/ Microdeletion within ESR1 constitutes a worldwide spread susceptibility factor for the development of undermasculinized genitalia HORMONE RESEARCH IN PAEDIATRICS
Prizes
[1]. 2024/10/ 学術賞
Memberships of academic societies
[1]. 日本小児科学会(評議員)
[2]. 日本内分泌学会
[3]. 日本小児内分泌学会(理事)
[4]. 生殖内分泌学会(評議員)
[5]. 日本遺伝学会
Committee Career
[1]. 2022/4/1- 日本小児内分泌学会 男女共同参画ワークライフバランス委員会
[2]. 2022/4/1- 日本小児内分泌学会 性分化・副腎
[3]. 2022/4/1- 日本小児内分泌学会 薬事委員会
[4]. 2022/4/1- 日本小児内分泌学会 渉外
Educational activities
Allotted class
[1]. 内分泌代謝
[2]. 小児医学
[3]. 小児科学
Social activities
Social Contribution
[1]. 2010/4/1- 託児者講習会